Chelation Therapy

You will find many forms of Chelation Therapy given by many levels of practitioners and following various formulas from the original three (3) hour drip to a 10 minute push. It is advised by Dr. Baum to choose a licensed D.O. or M.D. before considering such an invasive therapy due to possible side effects that are easily avoided by a medical doctor with the proper pharmaceutical background.

Our Albuquerque and Santa Fe Chelation Practice

Dr. Baum uses AST Chelation Therapy, using the ZETA Potential. This particular technique of administration was developed by T.C. McDaniel’s, D.O., from Cincinnati, Ohio, after treating thousands of patients over the past forty (40) years. Dr. McDaniel’s is noted by his fellow colleagues as a brilliant bio-chemist, instructor and clinician. He has authored two textbooks discussing the ZETA Potential and his technique. His principle method of treatment has proven very effective.
“Zeta Potential’s strength determines the amount of material that a fluid can carry. Increasing the electrical force in the solution, allows the fluid to dissolve and hold more material. In this way, deposits can be removed from many things.” T.C. McDaniel’s, D.O.

Dr. Baum’s current Chelation Therapy uses the ZETA Potential with an Intravenous administration that requires approximately one (1) hour in his office.

Oral Chelation Therapy

For those who want Oral Chelation, our office also carries Oral Chelation as prevention between IV Chelation Therapy treatments. Studies show that EDTA is not well enough absorbed from the intestinal tract into the blood stream to deal with the toxic metals and venerable athermanous plaque in the arterial vascular tree. Oral Chelation helps inhibit damage to the intestinal tract and arterial vascular system. It eliminates a high percentage of the toxic metal absorption and decreases the formation of free radicals. Oral Chelation is a good preventative but is not a substitute for Intravenous Chelation Therapy.

Introduction to Chelation Therapy

The use of Chelation therapy with intravenous ethylene diamine tetraacetate (EDTA) for the treatment of atherosclerosis is being used worldwide. Chelation Therapy began more than four decades ago and accelerates each year. Many published studies have confirmed the safety and effectiveness of intravenous EDTA for treatment of occlusive atherosclerotic arterial disease and age-related degenerative diseases. The scientific rationale of this therapy is the fact that it has been proven effective over and over again in clinical practice. Millions of patients have received more than thirty million infusions with no serious side effects when administered with the correct protocol. Research using laboratory animals has provided further support for the effectiveness of EDTA Chelation Therapy. There has never been a valid scientific study of EDTA Chelation that did not show its effectiveness. There have been some misleading unsupported medical reports with data that was erroneously interpreted as negative and present improper clinical validation.

What Does Chelation Mean?

Chelation (a work derived from the Greek work “chela” a claw is based on a simple fact that two or more attractive forces acting simultaneously on a metal atom are stronger than only one. A chelating agent is a molecule which contains at least two groups of polarity opposite to that of the atom it is wanted to remove, in such a sterical position as to fit the size of that atom, thus exerting a double or multiple pull on it. Where the atom is strongly anchored, more than two contact points may be needed.

Chelation Therapy and EDTA

The release of free radicals can be greatly reduced with the administration of EDTA in the use of Chelation Therapy. It is not possible for free radical reactions to be catalyzed and thus accelerated by metallic ions in the presence of EDTA. Traces of unbound metallic ions are necessary as catalysts of uncontrolled proliferation of free radicals in tissues. EDTA binds those ionic metals, making them chemically inert and rapidly removing them from the body.

Conditions Treated By Santa Fe AST Chelation Center

Toxic Metal Deposits, Blood Clots, High Blood Pressure, Circulatory Disorders, Arrhythmia, Peripheral Vascular Diseases, Chronic Fatigue, Fibromylagia, Treatment of Mercury Toxicity post Dental Amalgam removal, Dental Cavitations Infection Therapy.

Removing Toxic Heavy Metals with Chelation Therapy

EDTA was one of the first treatments for heavy metal poisoning and has been used successfully in its treatment for a long time. The process of Chelation is now used routinely for treatment of lead poisoning and for removal of radioactive bone-seeking elements such as plutonium. Toxic Heavy Metals are known to impede metabolism and promote degenerative disease processes in a variety of ways. Poisonous metals such as mercury, lead, nickel, and cadmium react avidly with sulfur-containing amino acids on protein molecules. When lead reacts with sulfur on the cysteine or methionine moiety of an enzyme, enzyme activity is reduced or destroyed. Lead is also known to displace zinc in zinc-dependent enzymes. Chelation Therapy re-activates enzymes by removing toxic metals. Since the industrial revolution the average concentration of lead in human bones has increased by nearly one thousand fold. The lead found in bones and other vital organs is released into our circulation with a fever and under stressful conditions increasing toxicity and causing more pain and suffering when it can least tolerate it. Mercury toxicity has not only increased with our consumption of fish over the last decade but with the old amalgam dental fillings that are slowly leaching into our system as they break down in our mouths. Mercury poisoning in the body is causing a multitude of pain syndromes, gallbladder and liver toxicity as well as heart palpitations.
Dr. Baum’s office performs an EDTA urine challenge test which is sent to a laboratory to determine the amount of heavy toxic metals pre-exist in your system prior to beginning IV Chelation Therapy treatments. These tests may also be run at intervals during the Chelation process to determine how effectiveness of the treatments.

What is Arthrosclerosis?

Any injury resulting in bleeding creates a homeostatic mechanism which quickly stops the flow of blood to prevent hemorrhage and death. Hormones, prostaglandins, fibrin and thromboplastin go into action to regulate blood loss under the control of a complex array of mechanisms. Prostaglandins are produced and degraded continuously as they have a half-life measured in seconds and must be constantly synthesized at a controlled rate with proper ratio between their various subtypes to maintain normal blood flow.

The two most important prostaglandins are prostacyclin and thromboxane. Prostacyclin reduces the adhesiveness of platelets, making it possible for blood and plasma to flow freely, reducing the tendency for fibrin deposition and thrombus formation. Prostacyclin relaxes muscle fibers in artery walls, thus reducing spasm. Thromboxane causes intense spasm in blood vessel walls and stimulates platelets to adhere creating the opposite effect. A proper balance between Prostacyclin and Thromboxane must be maintained to protect the artery against injury and hemorrhage and maintain proper circulation.

Free radicals and lipid peroxides greatly inhibit the synthesis of prostacyclin while thromboxane production remains unaffected. If lipid peroxides are present, either from dietary intake or hydrogenated fats and oils or from nearby per oxidation of lipid cell membranes, less prostacyclin is produced to balance the effects of thromboxane. Damage to the artery wall may also be caused by disordered immunity or bacteria. In the presence of good health these minor vascular injuries are rapidly healed by a layer of platelets coating the disrupted surface. Without adequate free radical protection the production of prostacyclin is blocked and without adequate prostacyclin, thromboxane is unopposed and causes the injured area of the arterial wall to attract platelets abnormally. When the production of platelets increases they begin to stick together which creates platelet aggregation. The growing layer of platelets traps leukocytes which in turn causes the production of more free radicals. This network of fibrin and microthrombi is formed and erythrocytes become trapped. Iron is released when some of the erythrocytes hemolyze and iron is released causing more free radical oxidation. Oxidizing any cholesterol present and damaging phospholipids in cell membranes prostacyclin production continues to be inhibited.

Cholesterol is oxidized by free radical activity and some of the cholesterol oxidation products ingested by atheroma cells have vitamin D activity. Vitamin D activity further assists the accumulation of calcium as plaques grow. Calcium begins to increase abnormally although calcium and cholesterol deposits in the arteries do not occur until late in the process or atheroma formation. Cholesterol which normally acts as a protection against further free radical damage becomes oxidized in the process and accumulates within the plaque. The plaque then gradually expands to exceed the blood supply and ulceration occurs.

This final process then contains plaque that degenerates into an amorphous fibro-fatty mass containing calcium, cholesterol, connective tissue and cellular debris. This necrotic core can rupture, releasing debris of plaque as more free radicals continue to suppress prostacyclin and further aggregation of platelets. Now platelets are releasing high concentrations of thromboxane and serotonin, leading to arterial spasm and ischemia.

Symptomatic ischemia usually does not occur until a blood vessel becomes at least 70 to 80 percent occluded. A meal laden with per oxidized fats can cause a sudden free radical insult, triggering an abrupt increase in spasm or even an acute thrombosis, superimposed on a partial occlusion, producing an infarction.

Cell damage can occur in any part of the body when cells swell and die as membranes become leaky and damaged. DNA change results in mutations, atheromata and even cancer. Lymphoid tissues and other cells of the immune system become damaged and tissues become stiffer and lose flexibility as cross-linkages occur in connective tissue, elastin and protein molecules. As tissues are damaged in this way they age more rapidly, organ functions deteriorate, joints become hypertrophic, inflamed and deformed with arthritis.

Selenium is inactivated by free radicals creating inert selenium compounds and causing metabolic selenium deficiency. Cancer patients excrete selenium in amounts up to five times the normal rate and selenium is just what they need the most.

Antigenic substances and malignant cells which would otherwise be neutralized can overwhelm a weakened immune system. Intact food molecules that leak cross the gut wall undigested are poorly tolerated and so called “food allergies” appear. Avoidance of sensitizing foods and other trigger factors becomes necessary to control symptoms. Candida albicans, yeast normally present in the body, then releases abnormal amounts of antigenic properties and toxins and can overwhelm the immune system attacking healthy tissues and leading to autoimmune syndromes.

If You Are Suffering From Any Of The Following Conditions

Cardiovascular Disease

Stomach Pain

Heart Burn

Chronic Diarrhea

Fatigue Syndrome

Fibromyalgia

Sarcoidosis

Wilson’s Syndrome

Hormonal Imbalance

CONTACT OUR CLINIC TODAY

Chelation Then and Now

Many years ago, there was concern of adverse effects on kidney function in normal functioning kidneys. This concern has proved unwarranted. Today the use of EDTA in different forms has been refined to a science that works if proper protocol is used in its administration. Please take the time to choose a licensed physician D.O. or M.D. who has the pharmaceutical knowledge to administer Chelation Therapy safely.

Prevention

Please visit the Diet For A Healthy Life section of our website or click:    Diet For A HealthyLife

AMA News article, why patients need fewer stents Click Here

You May Contact the office of James E. Baum, D.O. for further information about getting started: 505.989.8647 Santa Fe and 505.898.3354 Albuquerque

Note: Much of the above information was taken from articles and lectures given by two of the beginning founders of modern Chelation Therapy: Elmer M. Cranton, M.D. and Gary Gordon, D.O., M.D.

For More Information about IV Chelation Therapy or to find a Physician in your area: click here

BOOKS ON CHELATION THERAPY

A textbook on EDTA Chelation Therapy, edited by Elmer M. Cranton, M.D., forward by Linus Pauling, Ph.D
2. The Golden Years, by T.C. McDaniel’s, D.O.

Other Books Recommended by Dr. Baum
1. Eat Right For 4 Your Type, by Dr. Peter J. D’Adamo

Contact Us

SANTA FE OFFICE:
1850 Old Pecos Trail, Suite L
Santa Fe, NM 87505
505.989.8647 PHONE
505.983.6464 FAX
888.634.1492 TOLL-FREE